1. RAP can induce calcium overload in atrial myocyte.
快速心房起搏可导致细胞内钙离子超载。
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2. In this text, we reviewed the effect of atorvastatin on the treatment of cardiac myocyte hypertrophy.
本文从心肌细胞肥大这个角度阐述阿托伐他汀对心室重塑的影响。
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3. Objective: To study the current change of right ventricular myocyte ion channel after myocardial infarction.
目的探讨心肌梗死后右心室肌细胞离子通道电流的变化。
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4. Cell contraction and calcium transient are decreased in a single ventricular myocyte after CHF establishment.
心衰后心肌细胞收缩功能减弱，钙瞬变幅度减小。
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5. Results of ultrastructure of cardiac myocyte: Apoptosis could be observed in myocardial cell in diabetic group.
心肌细胞超微结构观察结果：糖尿病组心肌组织内可见凋亡的心肌细胞。
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6. METHODS: Patch clamp technique (whole cell recording) was used to record L-Ca2 + current in single cardiac myocyte.
方法：用全细胞膜片箝记录心室肌细胞的L -钙电流。
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7. Results The myocyte diameter and cross section were reduced progressively along with the duration of muscle denervation.
结果肌细胞直径及截面积随失神经支配时间延长呈进行性下降；
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8. Objective to construct a method of isolating single guinea pig atrial myocyte and to measure its electrophysiological properties.
目的建立单个心房肌细胞的急性分离方法并记录其电生理特性。
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9. Objective: to investigate the role of endoplasmic reticulum stress in induction of cardiac myocyte apoptosis in viral myocarditis.
目的：探讨内质网应激反应对于诱导病毒性心肌炎心肌细胞凋亡的作用。
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10. Myocyte enhancer factor2(MEF2) is an important factor in activating muscle-specific genes expression and controlling transcription.
肌细胞增强因子-2（M EF2A）是心肌细胞特异性基因表达和转录的中心调控因素。
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11. Objective: we studied the relationship between structural alteration of the myocyte and function under light and electron microscope.
目的：探讨法鲁四联症心肌细胞光电镜结构的变化与其功能的关系。
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12. Conclusion: These findings suggest endoplasmic reticulum stress may contribute to the cardiac myocyte apoptosis in viral myocarditis.
结论：内质网应激反应可能对于诱导病毒性心肌炎的心肌细胞凋亡有一定作用。
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13. Then we observe the change of myocardium enzymatic dynamics and detect myocyte apoptosis with electronic microscope and situ end labeling way.
观察各组家兔血清心肌酶学变化，并通过电镜形态学及原位末端标记法检测心肌细胞凋亡情况。
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14. Results:Cross section area of normal skeletal muscle fiber was large, morphous was regular, myocyte nucleus located in the edge of muscle fiber;
结果：正常骨骼肌纤维横截面积较大，形态规则，肌细胞核紧贴于肌纤维的边缘；
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15. The reduction rate of the myocyte diameter and the cross section in the right lower limb was slower significantly than that of the control side.
肌细胞直径及截面积，电刺激侧较对照侧肢体下降速度明显减慢；
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16. Methods Enzyme digestion was used to isolate single ventricular myocyte and whole cell patch clamp technique was used to record potassium current.
膜片钳技术是一种以记录离子通道的离子电流来反映细胞膜上单一的或多个的离子通道分子活动的技术。
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17. Objective to investigate the effect of HOE642 on cardiac myocyte apoptosis of the heterotopic heart transplantation of rat non heart-beating donors.
目的研究HOE642对热缺血死亡大鼠颈部异位停跳心脏供体心肌细胞凋亡的作用。
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18. Conclusion Myocyte apoptosis was directly induced by reperfution after ischemia, and apoptosis was a type of delayed cell death induced by reperfution.
结论再灌注直接导致了缺血后的心肌细胞凋亡，细胞凋亡是再灌注导致的迟发性心肌细胞死亡的一种方式。
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19. In traditional thoughts, apoptosis doesn't exist in the mature cells which doesn't have regenerative capacity, such as cardiac myocyte and nerve cells.
传统的观点认为细胞凋亡不发生于诸如成熟的心肌细胞、神经细胞等不具备再生能力的细胞中。
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20. Results: Research indicated that Leptin and Leptin receptor (OB-Rb) was closely related to the pathogenesis of Hypertensive Cardiac Myocyte Hypertrophy.
结果：研究表明瘦素和瘦素受体与高血压心肌肥厚的发病机制有密切关系。
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21. From a diagnostic standpoint, the aim is to try and develop biomarkers that identify patients with ACS, even when there is no evidence of myocyte necrosis.
从诊断的角度来看，其目标是即使在没有证据表明肌细胞坏死的情况下鉴别出acs的病人。
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22. This compensatory hypertrophy is pathologic because the process is maladaptive over time, with eventual myocyte loss, fibrosis, and ventricular dysfunction.
这种代偿性肥大随着时间的推移逐渐失去代偿，随着心肌细胞消失、纤维化，心室功能障碍。
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23. Objective: To investigate the relationships between the occurrence and the change of atrial myocyte structure and ultrastructure in atrial fibrillation(AF).
目的：探讨心房颤动（房颤）的发生、房颤转复后窦性心律的维持与相关细胞结构和超微结构变化的关系。
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24. Myocyte proliferation is positively correlated with heart systolic function, and negatively correlated with heart diastolic function in chronic heart failure.
心肌梗死大鼠心肌细胞增殖程度与心脏收缩功能呈正性相关，与舒张功能呈负性相关。
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25. Should myocardial ischemia continue too long, myocyte death occurs, and electrical shock after this will only result in pulseless electrical activity or asystole.
如果心肌缺血时间过长，心肌细胞死亡，此时的电除颤只会产生无脉性电活动或心搏停止。
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26. Methods Enzyme digestion was performed to isolate single ventricular myocyte of guinea pig and whole-cell patch-clamp technique was used to record potassium current.
27. In the article the effect of thyroid hormones was reviewed on cardiac ion channels and transports, so as to explain the changes of the action current on cardiac myocyte.
本文综述了甲状腺素对心脏离子通道和转运装置的作用，解释了其引起心肌细胞动作电位变化原因。
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28. After adjustment there were no statistically significant differences in survival between the Myocyte Necrosis and Clinical MI (low and high TnI) subgroups at any time point.
经过调整后，肌细胞坏死组和临床心肌梗塞组在任意时间点的存活率都没有明显统计学意义。
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29. As a result single guinea-pig atrial myocyte with normal electrophysiologic activity was obtained and its L-Ica and IK were successfully recorded with patch-clamp technique.
结果获得了具正常电生理活性的单个细胞及高阻封接的形成，成功记录了L -型钙流和延迟整流性钾流。
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30. As a result single guinea-pig atrial myocyte with normal electrophysiologic activity was obtained and its L-Ica and IK were successfully recorded with patch-clamp technique.
结果获得了具正常电生理活性的单个细胞及高阻封接的形成，成功记录了L -型钙流和延迟整流性钾流。
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用作名词 (n.)
Transmembrane ionic channel currents were recorded by using the whole-cell patchclamp technique in single cardiac entricular myocyte enzymatically isolated from guinea-pig hearts. 应用全细胞斑片钳技术记录酶学方法分离的豚鼠单个心室肌细胞离子通道电流。
